This is part 2 in a series of blog posts where I am sharing key highlights I took away from presentations, by Professor Cesar Fernandez de Las Penas, at the recent annual Spring Conference of the BMAS.
Part 1 can be viewed here...
With regards dry needling ‘mechanisms of action’ two main hypotheses were discussed:
Firstly a mechanical mechanism whereby the needle tip ‘destroys’ the dysfunctional motor end plate resulting in deactivation of the MTrP. This hypothesis is supported by evidence which shows a 60-70% decrease in local electrical activity within the end plate zone after dry needling. A certain amount of muscle damage has also be shown to occur, however 28 hours after dry needling the muscle morphology is restored to normal based on histological analysis. The normalisation of motor end plate activity after dry needling may partly explain a reduction in local muscle tone, amelioration of the local twitch response and improved muscle function.